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December online assessment

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1) A 55 year old man with Recurrent Focal Seizures Detailed patient case report here: http://ushaindurthi.blogspot.com/2020/11/55-year-old-male-with-complaints-of.html   1. What is the problem representation of this patient and what could be the anatomical site of lesion ?   ans:Problem statement Rights monoplegia of upperlimb-which is sudden onset with rt sided focal seizures with progression to secondary generalisation which was refractory to medical treatment for 2 days  uncontrolled sugars anatomical localisation of right sided focal seizures with secondary generalisation https://www.epilepsy.com/living-epilepsy/epilepsy-and/professional-health-care-providers/co-existing-disorders/brain-tumors-1 Anatomic location and General characteristics of seizures: Frontal lobe :  Usuallyoccur several times per day, short in duration, during sleep. Complex gestural automatisms common at onset. Tonic/postural manifestations prominent. Occipital lobe:Usually simple partial and secondarily genera
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 Internal assessment:  Antomocal Diagnosis: glomerulus of kidney Histological: ? Loss of effacement of foot process of podocytes of basement membrane ? Membranous glomerulopathy Etiological: diabetes and hypertension. Diabetic nephropathy    2.azotemia : Increased blood urea nitrogen with BUN to creat ratio 20:1 suggestive of renal or post renal azotemia. https://www.ncbi.nlm.nih.gov/books/NBK538145/   https://www.ncbi.nlm.nih.gov/books/NBK538145 Anemia:https://pubmed.ncbi.nlm.nih.gov/6876938/ https://spectrum.diabetesjournals.org/content/21/1/12 Hypoalbuminemia: Reduced protein intake and inflammation,due to reduced production of albumin by liver leading to decreased synthesis and increased catabolism and vascular permeability. https://pubmed.ncbi.nlm.nih.gov/9848794/ Acidosis: renal failure as there is decreased synthesis of bicarb resulting in high aniono gap metabolic acidosis. https://pubmed.ncbi.nlm.nih.gov/6876938/ 3.according to KDIGO guidelines,oral replacement therapy is init
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November online assessment

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 Question 1: https://sreejaboga.blogspot.com/2020/11/is-online-e-log-book-to-discuss-our.html?m=1 Epigastric pain anatomicaldanatomical  Inferior wall MI Pancreatitis  Duodenal ulcer Cholecystitis Causes of acute pancreatitis https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2665136 Pathophysiology of pancreatitis 2.sob: ARDS due to pancreatitis https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2864834/ ARDS secondary to sepsis Acidosis in renal failure https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6362877/ AKI management in pancreatitis Pleural effusion in pancreatitis. Question 2: https://aakansharaj.blogspot.com/2020/11/55-year-old-male-with-anemia.html?m=1l Bones  Patient has anemia,renal failure,lytic bony lesions,and pnemonia. Anemia is hypoproliferative picture Total protiens high 12.4and albumin 1.84( high gamma gap) MGUS premalignant condition with no organ failure Smoldering myeloma Anatomical locations of problems and etiologies: Bone marrow Lytic bony lesions of skull Anemia Renal fai
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